| Embolismo graso cerebral: un diagnóstico de reto. |
Cerebral Fat Embolism: A diagnostic challenge.
Gupta B, Kaur M, D'souza N, Dey CK, Shende S, Kumar A, Gamangatti S.
Department of Anaesthesia & Critical Care, JPNA Trauma Centre, All India Institute of Medical Sciences, New Delhi, India.
Saudi J Anaesth. 2011 Jul;5(3):348-52
Abstract
Fat embolism syndrome (FES) is a rare but a serious clinical catastrophe occurring after traumatic injury to long bones. Cerebral involvement in the absence of pulmonary or dermatological manifestation on initial presentation may delay the diagnosis of cerebral fat embolism (CFE). We discuss a case series of CFE which posed a challenge in diagnosis. The clinical presentations of these patients did not satisfy the commonly used clinical criteria for aiding the diagnosis of FES. Early MRI brain (DWI and T2 weighted sequences) in patients with neurological symptoms after trauma even in the absence of pulmonary and dermatological findings should be the goal.
http://www.saudija.org/text.asp?2011/5/3/348/84122
|
| Embolismo graso cerebral despues de remplazo bilateral total de rodilla. Informe de un caso. |
Cerebral fat embolism after bilateral total knee replacement arthroplasty -A case report-.
Chang RN, Kim JH, Lee H, Baik HJ, Chung RK, Kim CH, Hwang TH.
Department of Anesthesiology and Pain Medicine, College of Medicine, Ewha Womans University, Seoul, Korea.
Korean J Anesthesiol. 2010 Dec;59 Suppl:S207-10. Epub 2010 Dec 31.
Abstract
Fat embolism syndrome is a rare and potentially lethal complication most commonly seen in long bone fractures and intramedullary manipulation. The clinical triad of fat embolism syndrome consists of mental confusion, respiratory distress, and petechiae. This study reports a case of cerebral fat embolism syndrome following elective bilateral total knee replacement. After an uneventful anesthesia and initial recovery, the patient developed neurologic symptoms nine hours postoperatively
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3030038/pdf/kjae-59-S207.pdf
|
Embolismo graso cerebral sin cortocircuito intracardiaco. Un presentación nueva
|
Cerebral fat embolism without intracardiac shunt: A novel presentation.
Eriksson EA, Schultz SE, Cohle SD, Post KW.
Department of Trauma, Surgical Critical Care and Acute Care Surgery, Medical University of South Carolina, Charleston, SC, USA.
J Emerg Trauma Shock. 2011 Apr;4(2):309-12.
Abstract
Fat embolism syndrome (FES) is defined as an uncommon life-threatening disease process consisting of pulmonary, central nervous system (CNS), and cutaneous manifestations. The pathophysiology of this secondary injury is poorly understood. In the setting of the multiply injured patient, the diagnosis of FES is difficult to ascertain. A case report of a posttraumatic death caused by acute dissemination of diffuse fat emboli to the brain and lungs in the absence of a right-to-left heart defect after femur fracture is presented. The transesophageal echo cardiogram with bubble study failed to demonstrate an intracardiac defect or AV malformation in the lung further supporting a biochemical process. The acute decompensation of the patient within 2 h of the injury would favor mechanical emboli. Supportive care continues to be the mainstay of treatment for FES. Cerebral fat embolism should be considered in traumatically injured patients with unexplained decline in their neurologic examination. Cerebral fat embolism may occur without an intracardiac shunt.
http://www.onlinejets.org/text.asp?2011/4/2/309/82233
|
|
No hay comentarios:
Publicar un comentario